![]() ![]() In theory, the reasoning for treating subclinical hypothyroidism would be to decrease the risk of cardiovascular issues and potentially prevent it from progressing to overt hypothyroidism. Healthcare providers disagree on whether subclinical hypothyroidism needs to be treated due to conflicting studies showing its effectiveness. This leads to elevated TSH levels and normal thyroxine (T4) levels, resulting in subclinical hypothyroidism.ĭoes subclinical hypothyroidism need to be treated? However, in subclinical hypothyroidism, due to thyroid inflammation or other thyroid disease, thyroid hormonal output doesn’t increase like it normally should in response to the elevated TSH levels. ![]() When T4 and T3 levels drop, the cycle starts over again. These two hormones prevent your pituitary gland from producing more TSH if the levels of thyroxine and triiodothyronine are too high, thus completing the cycle. TSH then stimulates cells in your thyroid to release thyroxine or T4 (80%) and triiodothyronine or T3 (20%) into your bloodstream. To start, your hypothalamus releases thyroid-releasing hormone (TRH) to trigger the release of thyroid-stimulating hormone (TSH) by your pituitary gland. Normally, multiple hormones and glands in your endocrine system work together to carefully control the level of TSH in your bloodstream through a feedback loop. Diastolic hypertension ( high blood pressure).Being unable to tolerate cold temperatures.Depression and/or decreased attention span.However, it can sometimes present with mild symptoms of hypothyroidism, which include: Most of the time, subclinical hypothyroidism doesn’t cause any symptoms (it’s asymptomatic). What are the symptoms of subclinical hypothyroidism? It affects up to 10% of adults in the United States. How common is subclinical hypothyroidism? Most people with subclinical hypothyroidism in pregnancy won’t require treatment postpartum (after pregnancy). Pregnant people who have subclinical hypothyroidism and thyroid peroxidase (TPO) antibodies require thyroid replacement therapy (levothyroxine). Prior or current amiodarone or lithium use.History of preterm delivery, miscarriage and/or infertility.Type 1 diabetes and other autoimmune diseases.Personal or family history of thyroid disease.Healthcare providers typically only screen for subclinical hypothyroidism during pregnancy if you have risk factors for developing it, including: However, more recent studies have not replicated these associations. Impaired cognitive development in infants.Hypertensive disorders of pregnancy, including gestational high blood pressure and preeclampsia.Older studies have shown an association between subclinical hypothyroidism in pregnancy and the following conditions: It affects 15% to 28% of pregnant people.Įvidence linking subclinical hypothyroidism to issues during pregnancy is inconsistent and conflicting. Subclinical hypothyroidism is more common during pregnancy than overt hypothyroidism. Does subclinical hypothyroidism affect pregnancy? Who does subclinical hypothyroidism affect?Īnyone can have subclinical hypothyroidism, but it’s more likely to affect adults assigned female at birth and people over the age of 65. Subclinical hypothyroidism is often temporary but can be long-lasting. These two hormones are essential for maintaining your body’s metabolism - how your body transforms the food you eat into energy and uses it. Thyroid-stimulating hormone, commonly called TSH and also referred to as thyrotropin, is a hormone that your pituitary gland releases to trigger your thyroid to produce and release its own hormones - thyroxine (T4) and triiodothyronine (T3). “Subclinical” describes a condition that’s not severe enough to cause definite symptoms. Hypothyroidism happens when your thyroid doesn’t produce enough thyroid hormones (thyroxine and triiodothyronine). You don’t technically have hypothyroidism (commonly called overt hypothyroidism, in comparison), but it has the potential to develop into overt hypothyroidism. Subclinical hypothyroidism happens when you have elevated thyroid-stimulating hormone (TSH) levels with normal levels of thyroxine (T4). ![]()
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